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The Arabidopsis thaliana onset of leaf death 12 mutation in the lectin receptor kinase P2K2 results in an autoimmune phenotype

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Zhao,  L.
Transcription Factors and Gene Regulatory Networks, Cooperative Research Groups, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Mueller-Roeber,  B.
Transcription Factors and Gene Regulatory Networks, Cooperative Research Groups, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Citation

Zhao, L., Wang, H.-J., Martins, P. D., van Dongen, T., J., Bolger, A. M., et al. (2023). The Arabidopsis thaliana onset of leaf death 12 mutation in the lectin receptor kinase P2K2 results in an autoimmune phenotype. BMC Plant Biology, 23(1): 294. doi:10.1186/s12870-023-04300-0.


Cite as: https://hdl.handle.net/21.11116/0000-000D-3EFE-1
Abstract
Plant immunity relies on the perception of immunogenic signals by cell-surface and intracellular receptors and subsequent activation of defense responses like programmed cell death. Under certain circumstances, the fine-tuned innate immune system of plants results in the activation of autoimmune responses that cause constitutive defense responses and spontaneous cell death in the absence of pathogens.