Fructose induced KHK-C can increase ER stress independent of its effect on 
lipogenesis to drive liver disease in diet-induced and genetic models of NAFLD

Park, Se-Hyung; Helsley, Robert N.; Fadhul, Taghreed; Willoughby, Jennifer L.S.; Noetzli, Leila; Tu, Ho-Chou; Solheim, Marie Holm; Fujisaka, Shiho; Pan, Hui; Dreyfuss, Jonathan M.; Bons, Joanna; Rose, Jacob; King, Christina D.; Schilling, Birgit; Lusis, Aldons J.; Pan, Calvin; Gupta, Manoj; Kulkarni, Rohit N.; Fitzgerald, Kevin; Kern, Philip A.; Divanovic, Senad; Kahn, C. Ronald; Softic, Samir

doi:10.1016/j.metabol.2023.155591

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Fructose induced KHK-C can increase ER stress independent of its effect on lipogenesis to drive liver disease in diet-induced and genetic models of NAFLD

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Solheim, Marie Holm
Max Planck Institute for Metabolism Research, Brüning Group, Max Planck Society;

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Park, S.-H., Helsley, R. N., Fadhul, T., Willoughby, J. L., Noetzli, L., Tu, H.-C., et al. (2023). Fructose induced KHK-C can increase ER stress independent of its effect on lipogenesis to drive liver disease in diet-induced and genetic models of NAFLD. Metabolism-Clinical and Experimental, 145: 155591. doi:10.1016/j.metabol.2023.155591.

Cite as: https://hdl.handle.net/21.11116/0000-000D-6E17-F

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