Koenen, Michael Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;
https://link.springer.com/article/10.1007/s00424-023-02882-0 (Any fulltext)
https://link.springer.com/content/pdf/10.1007/s00424-023-02882-0.pdf (Any fulltext)
https://static-content.springer.com/esm/art%3A10.1007%2Fs00424-023-02882-0/MediaObjects/424_2023_2882_MOESM1_ESM.docx (Supplementary material)
Semino, F., Darche, F. F., Bruehl, C., Koenen, M., Skladny, H., Katus, H. A., et al. (2024). GPD1L-A306del modifies sodium current in a family carrying the dysfunctional SCN5A-G1661R mutation associated with Brugada syndrome. Pflügers Archiv: European Journal of Physiology, 476(2), 229-242. doi:10.1007/s00424-023-02882-0.