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The Evolutionary Genomics of Herbicide-Resistant Weeds

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Vasiljevic,  BW       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

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Lutz,  U       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

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Bezrukov,  I       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

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Weigel,  D       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

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Citation

Vasiljevic, B., Lutz, U., Bezrukov, I., & Weigel, D. (2020). The Evolutionary Genomics of Herbicide-Resistant Weeds. In Sixtieth Meeting of the Weed Science Society of America (WSSA 2020).


Cite as: https://hdl.handle.net/21.11116/0000-000E-1AEA-E
Abstract
The evolution of herbicide-resistant weeds is a predictable consequence of natural selection. Mechanisms underlying herbicide resistance are classified into two broad categories: the well-studied target-site-based resistance mechanisms (TSR), and non-target-site-based resistance mechanisms (NTSR) whose genetic determinants are poorly understood. Numerous studies have illustrated the quantitative nature of NTSR-associated traits. Owing to the high variability of NTSR, non- candidate-gene based approaches are necessary to elucidate the genetic basis underlying NTSR. In order to identify the natural modulators of glyphosate resistance, I am evaluating the differential response of over 100 Arabidopsis thaliana accessions to incremental doses of glyphosate for genome- wide association studies (GWAS). Preliminary analyses have hinted at a continuous phenotypic distribution suggesting the polygenic control of resistance. I will then conduct quantitative trait locus (QTL) mapping on segregating F2 populations derived from parental lines exhibiting extreme phenotypic responses. Combining both approaches will aid in resolving the complexity of genetic architectures underlying NTSR mechanisms and sets the stage for identifying causal loci in the evolution of NTSR after recurrent herbicide selection.