FIGNL1-FIRRM is essential for meiotic recombination and prevents DNA 
damage-independent RAD51 and DMC1 loading

Zainu, A.; Dupaigne, P.; Bouchouika, S.; Cau, J.; Clement, J. A. J.; Auffret, P.; Ropars, V.; Charbonnier, J.-B.; de Massy, B.; Mercier, R.; Kumar, R.; Baudat, F.

doi:10.1038/s41467-024-51458-8

Item

ITEM ACTIONSEXPORT

Add to Basket

Local TagsRelease HistoryDetailsSummary

Released

Journal Article

FIGNL1-FIRRM is essential for meiotic recombination and prevents DNA damage-independent RAD51 and DMC1 loading

MPS-Authors

/persons/resource/persons248362

Mercier, R.
Dept. of Chromosome Biology (Raphael Mercier), MPI for Plant Breeding Research, Max Planck Society;

External Resource

No external resources are shared

Fulltext (restricted access)

There are currently no full texts shared for your IP range.

Fulltext (public)

zainu_nat_comm_2024.pdf
(Publisher version), 5MB

Supplementary Material (public)

There is no public supplementary material available

Citation

Zainu, A., Dupaigne, P., Bouchouika, S., Cau, J., Clement, J. A. J., Auffret, P., et al. (2024). FIGNL1-FIRRM is essential for meiotic recombination and prevents DNA damage-independent RAD51 and DMC1 loading. Nature Communications, 15(1): 7015. doi:10.1038/s41467-024-51458-8.

Cite as: https://hdl.handle.net/21.11116/0000-0010-2154-A

Abstract

There is no abstract available