Insulin receptor signaling mediates APP processing and β-amyloid accumulation 
without altering survival in a transgenic mouse model of Alzheimer's disease

Stöhr, Oliver; Schilbach, Katharina; Moll, Lorna; Hettich, Moritz M.; Freude, Susanna; Wunderlich, Frank Thomas; Ernst, Marianne; Zemva, Johanna; Brüning, Jens Claus; Krone, Wilhelm; Udelhoven, Michael; Schubert, Markus

doi:10.1007/s11357-011-9333-2

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Insulin receptor signaling mediates APP processing and β-amyloid accumulation without altering survival in a transgenic mouse model of Alzheimer's disease

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Wunderlich, Frank Thomas
Wunderlich – Obesity and Cancer, Department Brüning, Max Planck Institute for Metabolism Research, Max Planck Society;

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Brüning, Jens Claus
Brüning – Neuronal Control of Metabolism, Department Brüning, Max Planck Institute for Metabolism Research, Max Planck Society;

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Stöhr, O., Schilbach, K., Moll, L., Hettich, M. M., Freude, S., Wunderlich, F. T., et al. (2013). Insulin receptor signaling mediates APP processing and β-amyloid accumulation without altering survival in a transgenic mouse model of Alzheimer's disease. AGE, 35(1), 83-101. doi:10.1007/s11357-011-9333-2.

Cite as: https://hdl.handle.net/11858/00-001M-0000-0026-D5FB-9