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Gpm6b deficiency impairs sensorimotor gating and modulates the behavioral response to a 5-HT2A/C receptor agonist

MPS-Authors
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Dere,  Ekrem
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Winkler,  Daniela
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Ritter,  Caroline
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Ronnenberg,  Anja
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Poggi,  Giulia
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Patzig,  Julia
Neurogenetics, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Nave,  Klaus-Armin
Neurogenetics, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Ehrenreich,  Hannelore
Clinical neuroscience, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Werner,  Hauke B.
Neurogenetics, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Citation

Dere, E., Winkler, D., Ritter, C., Ronnenberg, A., Poggi, G., Patzig, J., et al. (2015). Gpm6b deficiency impairs sensorimotor gating and modulates the behavioral response to a 5-HT2A/C receptor agonist. Behavioural Brain Research, 277, 254-263. doi:10.1016/j.bbr.2014.04.021.


Cite as: https://hdl.handle.net/11858/00-001M-0000-0029-70D1-B
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