Hyperekplexia phenotype of glycine receptor alpha 1 subunit mutant mice 
identifies Zn2+ as an essential endogenous modulator of glycinergic 
neurotransmission

Hirzel, K.; Müller, U.; Latal, A. T.; Hülsmann, S.; Grudzinska, J.; Seeliger, M. W.; Betz, H.; Laube, B.

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Hyperekplexia phenotype of glycine receptor alpha 1 subunit mutant mice identifies Zn2+ as an essential endogenous modulator of glycinergic neurotransmission

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Hirzel, K.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Müller, U.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Grudzinska, J.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Betz, H.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Laube, B.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Hirzel, K., Müller, U., Latal, A. T., Hülsmann, S., Grudzinska, J., Seeliger, M. W., et al. (2006). Hyperekplexia phenotype of glycine receptor alpha 1 subunit mutant mice identifies Zn2+ as an essential endogenous modulator of glycinergic neurotransmission. Neuron, 52(4), 679-690.

Cite as: https://hdl.handle.net/11858/00-001M-0000-002E-1E9D-3

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