date: 2021-12-06T18:02:00Z pdf:PDFVersion: 1.5 pdf:docinfo:title: Low-Expressing Synucleinopathy Mouse Models Based on Oligomer-Forming Mutations and C-Terminal Truncation of ?-Synuclein xmp:CreatorTool: LaTeX with hyperref package access_permission:can_print_degraded: true subject: ?-synuclein (?Syn) is the main protein component of Lewy bodies, intracellular inclusions found in the brain of Parkinson's disease (PD) patients. dc:format: application/pdf; version=1.5 pdf:docinfo:creator_tool: LaTeX with hyperref package access_permission:fill_in_form: true pdf:encrypted: false dc:title: Low-Expressing Synucleinopathy Mouse Models Based on Oligomer-Forming Mutations and C-Terminal Truncation of ?-Synuclein modified: 2021-12-06T18:02:00Z cp:subject: ?-synuclein (?Syn) is the main protein component of Lewy bodies, intracellular inclusions found in the brain of Parkinson's disease (PD) patients. pdf:docinfo:subject: ?-synuclein (?Syn) is the main protein component of Lewy bodies, intracellular inclusions found in the brain of Parkinson's disease (PD) patients. pdf:docinfo:creator: Gregor Eichele meta:author: Gregor Eichele meta:creation-date: 2021-06-14T12:51:38Z created: 2021-06-14T12:51:38Z access_permission:extract_for_accessibility: true Creation-Date: 2021-06-14T12:51:38Z Author: Gregor Eichele producer: pdfTeX-1.40.15 pdf:docinfo:producer: pdfTeX-1.40.15 pdf:unmappedUnicodeCharsPerPage: 6 dc:description: ?-synuclein (?Syn) is the main protein component of Lewy bodies, intracellular inclusions found in the brain of Parkinson's disease (PD) patients. Keywords: ?-Synuclein, Parkinson?s disease, transgenic mouse, truncation, oligomerization, motor impairment, neuroinflammation, olfaction deficiency access_permission:modify_annotations: true PDFVersion: 1.5 dc:creator: Gregor Eichele description: ?-synuclein (?Syn) is the main protein component of Lewy bodies, intracellular inclusions found in the brain of Parkinson's disease (PD) patients. dcterms:created: 2021-06-14T12:51:38Z Last-Modified: 2021-12-06T18:02:00Z dcterms:modified: 2021-12-06T18:02:00Z title: Low-Expressing Synucleinopathy Mouse Models Based on Oligomer-Forming Mutations and C-Terminal Truncation of ?-Synuclein xmpMM:DocumentID: uuid:5f2b1d18-8d7f-47f2-b139-05959b620541 Last-Save-Date: 2021-12-06T18:02:00Z pdf:docinfo:keywords: ?-Synuclein, Parkinson?s disease, transgenic mouse, truncation, oligomerization, motor impairment, neuroinflammation, olfaction deficiency pdf:docinfo:modified: 2021-12-06T18:02:00Z meta:save-date: 2021-12-06T18:02:00Z pdf:docinfo:custom:PDFVersion: 1.5 Content-Type: application/pdf X-Parsed-By: org.apache.tika.parser.DefaultParser creator: Gregor Eichele dc:subject: ?-Synuclein, Parkinson?s disease, transgenic mouse, truncation, oligomerization, motor impairment, neuroinflammation, olfaction deficiency access_permission:assemble_document: true xmpTPg:NPages: 17 pdf:charsPerPage: 3657 access_permission:extract_content: true access_permission:can_print: true meta:keyword: ?-Synuclein, Parkinson?s disease, transgenic mouse, truncation, oligomerization, motor impairment, neuroinflammation, olfaction deficiency access_permission:can_modify: true pdf:docinfo:created: 2021-06-14T12:51:38Z