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Tau mutant A152T, a risk factor for FTD/PSP, induces neuronal dysfunction and reduced lifespan independently of aggregation in a C. elegans Tauopathy model

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Pir,  Gulam Jeelani
Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Mandelkow,  Eckhard
Eckhard Mandelkow, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Mandelkow,  Eva-Maria
Eckhard Mandelkow, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Pir, G. J., Choudhary, B., Mandelkow, E., & Mandelkow, E.-M. (2016). Tau mutant A152T, a risk factor for FTD/PSP, induces neuronal dysfunction and reduced lifespan independently of aggregation in a C. elegans Tauopathy model. Molecular Neurodegeneration, 11(1): 96. doi:10.1186/s13024-016-0096-1.


Cite as: https://hdl.handle.net/11858/00-001M-0000-002A-70D3-9
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