English
 
Help Privacy Policy Disclaimer
  Advanced SearchBrowse
  1. View item / 
  2. Item Summary

Item

ITEM ACTIONSEXPORT
Add to Basket
  Local TagsRelease HistoryDetailsSummary

Released
Poster

Identification of Arabidopsis KUMONOSU gene involved in DNA methylation and heterochromatin-associated silencing

MPS-Authors
/persons/resource/persons271499

Becker,  C       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

/persons/resource/persons85266

Weigel,  D       
Department Molecular Biology, Max Planck Institute for Developmental Biology, Max Planck Society;

External Resource
No external resources are shared
Fulltext (restricted access)
There are currently no full texts shared for your IP range.
Fulltext (public)
There are no public fulltexts stored in PuRe
Supplementary Material (public)
There is no public supplementary material available
Citation

Ikeda, Y., Becker, C., López González, L., Pouch-Pélissier, M.-N., Pogorelnik, R., Weigel, D., et al. (2014). Identification of Arabidopsis KUMONOSU gene involved in DNA methylation and heterochromatin-associated silencing. Poster presented at FEBS EMBO 2014 Conference, Paris, France.


Cite as: https://hdl.handle.net/21.11116/0000-000F-37AF-F
Abstract
In plants, multiple layers of epigenetic regulators are required for heterochromatin-associated silencing, including many repetitive sequences and transposable elements (Regal and Mathieu, Bio- chim Biophys Acta., 2011). However, the control mechanism of tissue specific heterochromatic silencing is still unknown. Here, we report the new Arabidopsis gene affecting hetero- chromatin-associated silencing, KUMONOSU (KUN). We iso- lated kun mutant from the screening affecting transgene silencing. In kun mutant, not only transgene, many endogenous repetitive sequence and transposable elements were activated. Moreover, the expression of specific genic regions was also affected and DNA methylation level of many target genes was slightly decreased in the mutant. Interestingly, release of silencing of the reporter transgene was observed specifically in vascular tissues of kun mutant, which may indicate possible tissue-specificity in the regulation of heterochromatin silencing.