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Protein Kinase N Promotes Stress-Induced Cardiac Dysfunction Through Phosphorylation of Myocardin-Related Transcription Factor A and Disruption of Its Interaction With Actin

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Takefuji,  Mikito
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Wettschureck,  Nina
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Offermanns,  Stefan
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Sakaguchi, T., Takefuji, M., Wettschureck, N., Hamaguchi, T., Amano, M., Kato, K., Tsuda, T., Eguchi, S., Ishihama, S., Mori, Y., Yura, Y., Yoshida, T., Unno, K., Okumura, T., Ishii, H., Shimizu, Y., Bando, Y. K., Ohashi, K., Ouchi, N., Enomoto, A., Offermanns, S., Kaibuchi, K., & Murohara, T. (2019). Protein Kinase N Promotes Stress-Induced Cardiac Dysfunction Through Phosphorylation of Myocardin-Related Transcription Factor A and Disruption of Its Interaction With Actin. CIRCULATION, 140(21), 1737-1752. doi:10.1161/CIRCULATIONAHA.119.041019.


引用: https://hdl.handle.net/21.11116/0000-0006-AEE5-4
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